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The salvage pathway starts when the enzyme nicotinamide phosphoribosyltransferase (NAMPT) converts nicotinamide (NAM) to NMN
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The most common gross defect that promotes hyperuricemia in patients with gout without chronic kidney disease (CKD) is marked renal underexcretion of uric acid over broad ranges of serum urate levels ( Figure 1 - 7 )
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