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By targeting key molecular pathways such as Wnt/β-catenin, SMAD, PI3K/AKT, and MAPK/ERK, these therapies can modulate gene expression, cell signaling, and protein synthesis to optimize healing outcomes
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The evidence introduced thus far supports the scenario that AFB1 enhances the secretion of pro-inflammatory cytokines such as TNF-α and IL-6 in CNS-derived cells, leading to the promotion of immune responses and significant oxidative stress in the CNS (247)
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