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consequently, we need to overexpress SLC7A11 at higher levels to compete with other transporters for partnering with SLC3A2 in cells.) Notably, while moderate overexpression of SLC7A11 suppressed H 2 O 2 -induced cell death, as expected, high overexpression of SLC7A11 dramatically promoted cell death under H 2 O 2 treatment (Fig
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